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high glutathione circadian rhythm An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes glutathione S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in

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high glutathione circadian rhythm An integrative multi-omics analysis leveraging Mendelian randomization and  subsequent experimental validation prioritizes glutathione S-transferase mu  5 (GSTM5) as a genomic stability-related gene and a therapeutic  vulnerability to PLK1 inhibition in

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high glutathione circadian rhythm An integrative multi-omics analysis leveraging Mendelian randomization and  subsequent experimental validation prioritizes glutathione S-transferase mu  5 (GSTM5) as a genomic stability-related gene and a therapeutic  vulnerability to PLK1 inhibition in

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high glutathione circadian rhythm An integrative multi-omics analysis leveraging Mendelian randomization and  subsequent experimental validation prioritizes glutathione S-transferase mu  5 (GSTM5) as a genomic stability-related gene and a therapeutic  vulnerability to PLK1 inhibition in
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